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AGN-241751

Syndeio Biosciences, Inc · Phase 2 active Small molecule

AGN-241751 is a Sodium-activated potassium channel inhibitor Small molecule drug developed by Syndeio Biosciences, Inc. It is currently in Phase 2 development for Atrial fibrillation for stroke prevention.

AGN-241751 is a small molecule inhibitor of the sodium-activated potassium channel, Nav1.5.

AGN-241751 is a small molecule inhibitor of the sodium-activated potassium channel, Nav1.5. Used for Atrial fibrillation for stroke prevention.

Likelihood of approval
13.3% vs 15.3% industry baseline
If approved by FDA: likely 2031–2034
Steps remaining: Phase 3 → NDA/BLA submission
Confidence: Medium
Why this estimate
  • Baseline phase 2 → approval rate +15.3pp
    Industry-wide phase 2 drugs reach approval ~15.3% of the time (BIO/Informa 2023 industry benchmark across all therapeutic areas).
  • Cardiovascular Phase 3 risk -2.0pp
    Modern cardiovascular outcome trials are large + long; many fail to beat aggressive standard-of-care.
Predicted approval windows by jurisdiction (conditional on FDA approval)
Regulator Country Likely year Lag vs FDA
FDA US 2031–2034
EMA EU 2032–2035 +0.7 yr
MHRA GB 2032–2035 +0.7 yr
Health Canada CA 2032–2036 +0.9 yr
TGA AU 2032–2036 +1.2 yr
PMDA JP 2032–2036 +1.5 yr
NMPA CN 2033–2037 +2.3 yr
MFDS KR 2032–2036 +1.4 yr
CDSCO IN 2032–2037 +1.8 yr
ANVISA BR 2033–2037 +2.3 yr

Hover any row for the lag rationale. Lag estimates are reduced when the drug has FDA Breakthrough or EMA PRIME designation (sponsors file globally in parallel).

Estimate based on the BIO/Informa industry phase transition rates plus per-drug modifiers for therapeutic area, sponsor type, FDA designations, mechanism, and trial design. Per-jurisdiction lags from Tufts CSDD international approval studies. Not investment, clinical or regulatory advice. Methodology: /methodology#likelihood.

At a glance

Generic nameAGN-241751
SponsorSyndeio Biosciences, Inc
Drug classSodium-activated potassium channel inhibitor
TargetNav1.5
ModalitySmall molecule
Therapeutic areaCardiovascular
PhasePhase 2

Mechanism of action

By blocking Nav1.5, AGN-241751 aims to reduce the abnormal electrical activity in the heart that can lead to atrial fibrillation. This mechanism is thought to be beneficial for patients with atrial fibrillation who are at risk of stroke.

Approved indications

Common side effects

Key clinical trials

Primary sources

Every claim on this page is sourced from regulatory or scientific primary sources. See our editorial policy for full methodology.

SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

Competitive intelligence

For the full competitive landscape — auto-detected comparators, recent regulatory actions across the set, upcoming PDUFA, patent timeline, sponsor landscape:

Frequently asked questions about AGN-241751

What is AGN-241751?

AGN-241751 is a Sodium-activated potassium channel inhibitor drug developed by Syndeio Biosciences, Inc, indicated for Atrial fibrillation for stroke prevention.

How does AGN-241751 work?

AGN-241751 is a small molecule inhibitor of the sodium-activated potassium channel, Nav1.5.

What is AGN-241751 used for?

AGN-241751 is indicated for Atrial fibrillation for stroke prevention.

Who makes AGN-241751?

AGN-241751 is developed by Syndeio Biosciences, Inc (see full Syndeio Biosciences, Inc pipeline at /company/syndeio-biosciences-inc).

What drug class is AGN-241751 in?

AGN-241751 belongs to the Sodium-activated potassium channel inhibitor class. See all Sodium-activated potassium channel inhibitor drugs at /class/sodium-activated-potassium-channel-inhibitor.

What development phase is AGN-241751 in?

AGN-241751 is in Phase 2.

What are the side effects of AGN-241751?

Common side effects of AGN-241751 include Increased risk of atrial fibrillation.

What does AGN-241751 target?

AGN-241751 targets Nav1.5 and is a Sodium-activated potassium channel inhibitor.

Related

Primary sources · FDA · ClinicalTrials.gov · EMA · SEC EDGAR · ChEMBL · Wikidata · full sourcing