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Acetylsalicylic Acid (ASA)

Sunnybrook Health Sciences Centre · FDA-approved active Small molecule

Acetylsalicylic acid irreversibly inhibits cyclooxygenase (COX) enzymes, reducing the production of prostaglandins and thromboxane to decrease inflammation, pain, and platelet aggregation.

Acetylsalicylic acid irreversibly inhibits cyclooxygenase (COX) enzymes, reducing the production of prostaglandins and thromboxane to decrease inflammation, pain, and platelet aggregation. Used for Acute myocardial infarction prevention and secondary prevention, Ischemic stroke and transient ischemic attack prevention, Unstable angina.

At a glance

Generic nameAcetylsalicylic Acid (ASA)
Also known asAspirin, aspirin, Chewable aspirin
SponsorSunnybrook Health Sciences Centre
Drug classNonsteroidal anti-inflammatory drug (NSAID); antiplatelet agent
TargetCyclooxygenase-1 (COX-1); Cyclooxygenase-2 (COX-2)
ModalitySmall molecule
Therapeutic areaCardiovascular; Pain management; Inflammation
PhaseFDA-approved

Mechanism of action

ASA acetylates serine residues on COX-1 and COX-2 enzymes, permanently blocking their catalytic activity. This prevents the synthesis of prostaglandins (which mediate inflammation and pain) and thromboxane A2 (which promotes platelet aggregation). The antiplatelet effect is particularly important at low doses, where COX-1 inhibition in platelets predominates, making ASA useful for cardiovascular protection.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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