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NCT06890780

Observation on the Correlation Between Serum/Fecal Isoflavones, Abundance of TMA-producing Bacteria and Serum TMAO in Hyperlipidemia and Healthy Subjects

Recruiting now Last updated 24 March 2025
What this trial tests

trial in Hyperlipidemia in 200 participants. Currently enrolling.

Timeline
1 January 2023
Primary endpoint
1 January 2024
31 December 2025

Quick facts

Lead sponsorZhujiang Hospital
StatusRecruiting now
Study typeOBSERVATIONAL
Enrollment200
Start date1 January 2023
Primary completion1 January 2024
Estimated completion31 December 2025
Sites1 location across China

Conditions studied

Sponsor

Zhujiang Hospital

Who can join

Adults 18 to 70, any sex, with Hyperlipidemia. Patients with the condition only — healthy volunteers not accepted.

Sponsor's own description

Previous studies have shown that trimethylamine N-oxide (TMAO), a gut microbiota-related metabolite, plays a significant role in the development and progression of cardiovascular disease (CVD). How to regulate the structure of gut microbiota to reduce circulating TMAO levels in the host is currently one of the hot topics in research. Diet is a major factor shaping the structure of gut microbiota. Through the exploration of dietary elements, we have found that multiple epidemiological studies suggest an inverse correlation between the intake of isoflavones and CVD, indicating that isoflavones are potential agents for the prevention and treatment of CVD. Interestingly, isoflavones have poor water solubility and low bioavailability. Several studies have confirmed interactions between isoflavones and gut microbiota, suggesting that the gut and gut microbiota are likely important therapeutic targets for isoflavones in preventing and treating CVD. Furthermore, a high-fat diet (HFD) is also an independent risk factor for CVD. Research literature indicates that HFD can disrupt both the gut and gut microbiota. As a biomarker for CVD risk, TMAO has been reported in some studies to increase in circulation following HFD intake, but the mechanisms behind this phenomenon require further exploration. Based on the above literature, we propose a scientific hypothesis: Can isoflavones regulate gut microbiota and subsequently reduce serum TMAO levels in HFD-fed mice? This hypothesis can be further divided into three specific scientific questions: Which isoflavones can reduce serum TMAO levels in HFD-fed mice? Is gut microbiota the key factor through which isoflavones reduce serum TMAO levels in HFD-fed mice? What mechanisms do these substances use to modulate gut microbiota?

Publications & conference data

No peer-reviewed publications indexed yet for this trial.

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Other recruiting trials for Hyperlipidemia

Currently open trials in the same condition.

Other Zhujiang Hospital trials

Trials by the same sponsor.

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Data sources for this page

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