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NCT05630989

A Registry to Capture Patient Outcomes With KRAS G12R Altered Advanced Pancreatic Ductal Adenocarcinoma Treated With MEK Inhibitor-based Combination Therapy

Recruiting now Last updated 24 April 2025
What this trial tests

trial testing combination therapy with no MEKi in Pancreatic Ductal Adenocarcinoma in 80 participants. Currently enrolling.

Timeline
7 February 2023
Primary endpoint
1 August 2027
1 August 2027

Quick facts

Lead sponsorMandana Kamgar, MD
StatusRecruiting now
Study typeOBSERVATIONAL
Enrollment80
Start date7 February 2023
Primary completion1 August 2027
Estimated completion1 August 2027
Sites1 location across United States

Drugs / interventions tested

Conditions studied

Sponsor

Mandana Kamgar, MD — full company profile →

Who can join

18 and older, any sex, with Pancreatic Ductal Adenocarcinoma. Patients with the condition only — healthy volunteers not accepted.

Sponsor's own description

This is an observational precision oncology study designed to collect and analyze data that allows us to characterize the safety and efficacy of several different mitogen-activated protein kinase kinase inhibitor (MEKi) -based treatment strategies and the feasibility of administering MEKi combination therapies to patients with KRAS G12R mutated advanced pancreatic ductal adenocarcinoma (PDAC).

Publications & conference data

4 peer-reviewed publications reference this trial (live from Europe PMC):

  1. Genetics and biology of pancreatic ductal adenocarcinoma.
    Ying H, Kimmelman AC, Bardeesy N, Kalluri R, et al · · 2025 · cited 20× · PMID 39510840 · DOI 10.1101/gad.351863.124
  2. Kinome state is predictive of cell viability in pancreatic cancer tumor and cancer-associated fibroblast cell lines.
    Berginski ME, Jenner MR, Joisa CU, Herrera Loeza G, et al · · 2024 · cited 3× · PMID 39221276 · DOI 10.7717/peerj.17797
  3. Decoding Drug Resistance in Pancreatic Cancer: A Subcellular Structure Perspective.
    Li X, Lyu H, Wu Y, Chen A, et al · · 2026 · PMID 41972577 · DOI 10.3390/biology15070574
  4. Mutant and Wild-Type RAS Cross-talk and Stoichiometric Deficiencies Are Determinants of Sensitivity to Targeted Therapies in KRASG12R Pancreatic Ductal Adenocarcinoma.
    Kamgar M, Hobbes GA, Davidson R, Dorbin D, et al · · 2026 · PMID 41134578 · DOI 10.1158/0008-5472.can-25-0018

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Other recruiting trials for Pancreatic Ductal Adenocarcinoma

Currently open trials in the same condition.

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Drug Landscape aggregates and links these public records for informational use only. Always verify against the primary source before clinical or regulatory decisions. Canonical URL: https://druglandscape.com/trial/NCT05630989.

Primary sources · FDA · ClinicalTrials.gov · EMA · SEC EDGAR · ChEMBL · Wikidata · full sourcing