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NCT05372458

The Mechanism Study of Diabetic Pancreatic Amyloid Deposition on Cognitive Dysfunction in Alzheimer's Disease

Status unknown Last updated 12 May 2022
What this trial tests

trial in AD in 400 participants. Status unknown.

Timeline
1 January 2021
Primary endpoint
31 December 2023
31 December 2023

Quick facts

Lead sponsorHuashan Hospital
StatusStatus unknown
Study typeOBSERVATIONAL
Enrollment400
Start date1 January 2021
Primary completion31 December 2023
Estimated completion31 December 2023
Sites1 location across China

Conditions studied

Sponsor

Huashan Hospital

Who can join

Adults 60 to 80, any sex, with AD or DM. Patients with the condition only — healthy volunteers not accepted.

Sponsor's own description

Alzheimer's disease (AD) is closely related to diabetes (DM). DM will aggravate the progression of AD, but the specific mechanism has not yet been clarified. Previous study found that a key pathological feature of the pancreas in patients with DM is islet amyloid polypeptide, and there is also islet amyloid polypeptide in the brain. Therefore, DM may cause cytotoxicity through the interaction between pancreatic amyloid and brain Aβ protein and further aggravate AD progress. In this study, starting with DM and AD pathological biomarkers, the amyloid PET target molecular probe 18F-AV45 will be used to monitor the dynamic changes of amyloid protein in the brain and pancreas during the development of AD. The completion of this study will provide a new view for understanding the mechanism of DM on AD cognitive dysfunction and effectively preventing and treating these two diseases.

Publications & conference data

No peer-reviewed publications indexed yet for this trial.

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Other recruiting trials for AD

Currently open trials in the same condition.

Other Huashan Hospital trials

Trials by the same sponsor.

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Data sources for this page

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