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NCT05225870

Significance of Cortactin Expression in Colorectal Adenocarcinomas

Completed NA Last updated 15 July 2022
What this trial tests

NA trial testing immunohistochemical staining in Cancer Colon in 50 participants. Completed in 30 December 2021.

Timeline
1 January 2021
Primary endpoint
30 September 2021
30 December 2021

Quick facts

Lead sponsorSohag University
PhaseNA
StatusCompleted
Study typeINTERVENTIONAL
Allocationna
Designsingle group
Maskingnone
Primary purposebasic science
Enrollment50
Start date1 January 2021
Primary completion30 September 2021
Estimated completion30 December 2021
Sites1 location across Egypt

Drugs / interventions tested

Conditions studied

Sponsor

Sohag University

Who can join

Adults 30 to 80, any sex, with Cancer Colon. Patients with the condition only — healthy volunteers not accepted.

Sponsor's own description

Colorectal carcinoma is one of the most aggressive malignant epithelial neoplasms affecting the gastrointestinal tract. The incidence of colorectal carcinoma is obviously increasing in developing countries, where the physical inactivity and the consumption of animal fat-rich food became more evident. Colorectal tumorigenesis is a multistep process which is initiated by adenoma and is terminated by carcinoma, the latter shows variable degrees of tumor differentiation and invasiveness. During the adenoma-carcinoma process; a series of genetic mutations occur. Detection of these genetic mutations will help in the development of novel therapeutic agents, which in turn will improve patients' outcomes. Cortactin (CTTN) is a Src kinase substrate, encoded by a gene located on chromosome 11. CTTN binds to and activates Arp 2/3 and stabilizes the dynamic actin assembly after its formation. So, it become clear that CTTN is involved in the formation of the leading-edges cellular protrusions.

Publications & conference data

No peer-reviewed publications indexed yet for this trial. Completed trials usually publish results within 12-18 months.

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Other Sohag University trials

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Data sources for this page

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