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NCT04572477

The Influence of Sequential Tau Protein and Amyloid Plaque Imaging Changes on Stroke Prognosis and Cognitive Outcome

Status unknown Phase 2 Last updated 6 May 2023
What this trial tests

Phase 2 trial testing [18F]THK-5351 in Post-stroke Dementia, Vascular Mild Cognitive Impairment in 200 participants. Status unknown.

Timeline
1 June 2017
Primary endpoint
30 November 2023
31 May 2024

Quick facts

Lead sponsorChang Gung Memorial Hospital
PhasePhase 2
StatusStatus unknown
Study typeINTERVENTIONAL
Allocationnon randomized
Designparallel
Maskingsingle
Primary purposediagnostic
Enrollment200
Start date1 June 2017
Primary completion30 November 2023
Estimated completion31 May 2024
Sites1 location across Taiwan

Drugs / interventions tested

Conditions studied

Sponsor

Chang Gung Memorial Hospital

Who can join

Adults 50 to 85, any sex, with Post-stroke Dementia, Vascular Mild Cognitive Impairment. Patients with the condition only — healthy volunteers not accepted.

Sponsor's own description

Amyloid plaques and tau protein are the landmarks of neurodegeneration in Alzheimer's disease (AD). On the other hand, it is reported that cerebral ischemia may induce amyloid plaques and tau protein accumulation. However, it was difficult to in vivo disentangle the complex and dynamic interactions between AD pathophysiology and cerebral vascular injury in the development of post-stroke cognitive impairment in the past. With the advent of novel radiotracers specific to cerebral amyloid plaques and tau protein, we aim to conduct a prospective multimodal neuroimaging cohort study to investigate the contribution of vascular injury, amyloid plaques and tau protein to stroke recovery and post-stroke cognitive impairment.

Publications & conference data

No peer-reviewed publications indexed yet for this trial.

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Other trials of [18F]THK-5351

Trials testing the same drug.

Other Chang Gung Memorial Hospital trials

Trials by the same sponsor.

Verify against primary sources

Data sources for this page

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