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NCT04116242

MERTK Signalling in Monocytes/Macrophages in Patients With Liver Disease

Completed Last updated 25 June 2024
What this trial tests

trial testing blood sampling for research purpose in Liver Disease in 277 participants. Completed in 10 June 2024.

Timeline
27 August 2015
Primary endpoint
10 June 2024
10 June 2024

Quick facts

Lead sponsorUniversity Hospital, Basel, Switzerland
StatusCompleted
Study typeOBSERVATIONAL
Enrollment277
Start date27 August 2015
Primary completion10 June 2024
Estimated completion10 June 2024
Sites4 locations across United Kingdom, Switzerland

Drugs / interventions tested

Conditions studied

Sponsor

University Hospital, Basel, Switzerland

Who can join

18 and older, any sex, with Liver Disease or Cirrhosis of the Liver. Patients with the condition only — healthy volunteers not accepted.

Sponsor's own description

This study is to investigate MER receptor tyrosine kinase (MERTK) signalling cascade on monocytes and tissue macrophages in respect to innate immune function of the cells in patients with cirrhosis at different stages of disease (Child A, B, C, acute decompensation, acute-on-chronic liver failure (ACLF)) and in comparison to patients with acute liver failure and to healthy controls.

Publications & conference data

2 peer-reviewed publications reference this trial (live from Europe PMC):

  1. Expression of AXL receptor tyrosine kinase relates to monocyte dysfunction and severity of cirrhosis.
    Brenig R, Pop OT, Triantafyllou E, Geng A, et al · · 2020 · cited 34× · PMID 31822557 · DOI 10.26508/lsa.201900465
  2. AXL Expression on Homeostatic Resident Liver Macrophages Is Reduced in Cirrhosis Following GAS6 Production by Hepatic Stellate Cells.
    Pop OT, Geng A, Flint E, Singanayagam A, et al · · 2023 · cited 15× · PMID 37004869 · DOI 10.1016/j.jcmgh.2023.03.007

Verify or expand the search:

Other recruiting trials for Liver Disease

Currently open trials in the same condition.

Other University Hospital, Basel, Switzerland trials

Trials by the same sponsor.

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Data sources for this page

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