Last reviewed · How we verify

NCT03591939

T-regulatory Cells in Diabetic Type Two Nephropathy

Status unknown Last updated 19 July 2018
What this trial tests

trial testing laboratory test in Diabetic Nephropathy Type 2 in 63 participants. Status unknown.

Timeline
1 August 2018
Primary endpoint
1 August 2019
1 October 2019

Quick facts

Lead sponsorAssiut University
StatusStatus unknown
Study typeOBSERVATIONAL
Enrollment63
Start date1 August 2018
Primary completion1 August 2019
Estimated completion1 October 2019

Drugs / interventions tested

Conditions studied

Sponsor

Assiut University

Who can join

Adults 30 to 70, any sex, with Diabetic Nephropathy Type 2. Patients with the condition only — healthy volunteers not accepted.

Sponsor's own description

Diabetes mellitus is one of the most prevalent health problems worldwide. Diabetic nephropathy has become the leading cause of end-stage kidney disease worldwide and is associated with an increased cardiovascular risk. Traditionally, metabolic and hemodynamic factors are the main causes of renal lesions in patients with type two diabetes mellitus and diabetic nephropathy , both considered non-immune diseases. Serial researches has demonstrated that diabetic nephropathy is a metabolic and hemodynamic disorder, with inflammation playing a vital role in the process.

Publications & conference data

No peer-reviewed publications indexed yet for this trial.

Verify or expand the search:

Other trials of laboratory test

Trials testing the same drug.

Other recruiting trials for Diabetic Nephropathy Type 2

Currently open trials in the same condition.

Other Assiut University trials

Trials by the same sponsor.

Verify against primary sources

Data sources for this page

Drug Landscape aggregates and links these public records for informational use only. Always verify against the primary source before clinical or regulatory decisions. Canonical URL: https://druglandscape.com/trial/NCT03591939.

Primary sources · FDA · ClinicalTrials.gov · EMA · SEC EDGAR · ChEMBL · Wikidata · full sourcing