Last reviewed · How we verify

SGLT2 Inhibition and Left Ventricular Mass (EMPATROPHY)

NCT02728453 PHASE4 TERMINATED

Patients with type 2 diabetes mellitus are exposed to an excessive heart failure risk secondary to left ventricular hypertrophy and impaired diastolic filling, a condition not addressed by currently available treatments. The abnormality results from obesity-induced volume overload, increased blood pressure, and myocardial fat accumulation. By improving metabolism, body weight, and blood pressure, Empagliflozin addresses the root causes of type 2 diabetes-associated myocardial disease. We will assess left ventricular mass, function, and lipid content in patients with type 2 diabetes mellitus using cardiac magnetic resonance imaging and spectroscopy as well as echocardiography before and after empagliflozin or glimepiride treatment. We expect to observe improvements in left ventricular mass, function, and fat content with empagliflozin. The results of the study will help to position empagliflozin as an antidiabetic agent with the added value of protecting the heart.

Details

Lead sponsorHannover Medical School
PhasePHASE4
StatusTERMINATED
Enrolment7
Start dateWed Apr 27 2016 00:00:00 GMT+0000 (Coordinated Universal Time)
CompletionWed Sep 27 2017 00:00:00 GMT+0000 (Coordinated Universal Time)

Conditions

Interventions

Countries

Germany