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NCT02441283

A Study to Assess Resistance and Durability of Response to ABT-493 and/or ABT-530

Completed Phase 2, PHASE3 Results posted Last updated 21 September 2020
What this trial tests

Phase 2, PHASE3 trial testing ABT-493 in Hepatitis C in 384 participants. Completed in 15 October 2019.

Timeline
22 June 2015
Primary endpoint
15 October 2019
15 October 2019

Quick facts

Lead sponsorAbbVie
PhasePhase 2, PHASE3
StatusCompleted
Study typeINTERVENTIONAL
Allocationna
Designsingle group
Maskingnone
Primary purposeother
Enrollment384
Start date22 June 2015
Primary completion15 October 2019
Estimated completion15 October 2019
Sites42 locations across New Zealand, Belgium, United Kingdom, Germany, Canada, Puerto Rico, Australia, United States

Drugs / interventions tested

Conditions studied

Sponsor

AbbVie — full company profile →

Who can join

Adults 18 to 80, any sex, with Hepatitis C. Patients with the condition only — healthy volunteers not accepted.

Results — posted to ClinicalTrials.gov

Per-arm endpoint measurements with 95% confidence intervals where reported. Source: trial results section.

Percentage of Participants Who Maintained a Sustained Virologic Response (SVR) Out of Those Who Achieved SVR12 in a Prior Study With an ABT-493- and/or ABT-530-containing Regimen Primary · From the end of treatment in the previous study up to 3 years post-treatment

Maintaining a sustained virologic response (SVR) is defined as having Hepatitis C virus ribonucleic acid (HCV RNA) value consistently below the lower limit of quantification (\< LLOQ) from the end of treatment in the previous study throughout Study M13-576 (this study).

GroupValue95% CI
HCV-infected Participants99.5
Percentage of Participants Who Relapsed or Had a New Hepatitis C Virus (HCV) Infection at Any Time up to the Last Follow-up in This Study Among Participants Who Achieved SVR12 in a Prior Study With an ABT-493- and/or ABT-530-containing Regimen Primary · From the end of treatment in the previous study up to 3 years post-treatment

Relapse was defined as confirmed Hepatitis C virus ribonucleic acid (HCV RNA) greater than or equal to the lower limit of quantification (≥ LLOQ) between end of treatment and up to and including the last HCV RNA measurement collected in this study for a participant with HCV RNA \< LLOQ at Final Treatment Visit who completed treatment, excluding reinfection. HCV reinfection was defined as confirmed HCV RNA ≥ LLOQ after the end of treatment in a participant who had HCV RNA \< LLOQ at Final Treatment Visit, along with the post-treatment detection of a different HCV genotype, subtype, or clade com

Relapse overall
GroupValue95% CI
HCV-infected Participants0.3
Reinfection overall
GroupValue95% CI
HCV-infected Participants0.3
Number of Participants With Persistence of Resistance-Associated Amino Acid Variants Among Those Experiencing Virologic Failure Primary · From Day 1 to Month 12

Plasma samples for HCV resistance testing were collected at study visits. The variants in nonstructural viral protein 3 (NS3) and nonstructural viral protein 5A (NS5A) at amino acid positions of interest were analyzed by next generation sequencing relative to baseline and prototypic reference sequences.

NS3 Variants at Months 3, 6, and 12
GroupValue95% CI
HCV-infected Participants1
NS5A Variants at Months 3, 6, and 12
GroupValue95% CI
HCV-infected Participants1
Number of Participants With Medical Events Related to Progression of Liver Disease or Hepatitis C Virus Infection Secondary · After Day 1 up to 3 years post-treatment

Any events (i.e., diagnoses) related to liver disease progression and/or HCV infection considered to be clinically significant by the investigator that began or worsened after Day 1 were documented until the end of the study or initiation of re-treatment for HCV (if applicable). Significant events related to liver disease progression include the development of cirrhosis, events indicative of hepatic decompensation, (including: variceal bleeding, new ascites, spontaneous bacterial peritonitis, hepatic encephalopathy, hepato-renal syndrome, hepatic hydrothorax or other evidence of hepatic decomp

Medical events related to liver disease or HCV inf
GroupValue95% CI
HCV-infected Participants7
Development of cirrhosis
GroupValue95% CI
HCV-infected Participants0
Liver decompensation (Variceal bleeding)
GroupValue95% CI
HCV-infected Participants0
Liver decompensation (Ascites)
GroupValue95% CI
HCV-infected Participants0
Liver decomp (spontaneous bacterial peritonitis)
GroupValue95% CI
HCV-infected Participants0
Liver decompensation (hepatic encephalopathy)
GroupValue95% CI
HCV-infected Participants0
Liver decompensation (hepatorenal syndrome)
GroupValue95% CI
HCV-infected Participants0
Liver decompensation (hepatic hydrothorax)
GroupValue95% CI
HCV-infected Participants0
Mean Concentration of Interferon Gamma-induced Protein 10 (IP-10) Over Time Secondary · From Day 1 up to 3 years post-treatment

A plasma sample for IP-10 testing was collected at study visits for all participants, until the end of the study or initiation of re-treatment for HCV (if applicable). IP-10 is used to assess liver fibrosis in participants with chronic liver disease.

Day 1
GroupValue95% CI
HCV-infected Participants146.884± 116.6272
Month 3
GroupValue95% CI
HCV-infected Participants147.606± 118.5143
Month 6
GroupValue95% CI
HCV-infected Participants148.959± 119.0047
Month 12
GroupValue95% CI
HCV-infected Participants224.400
Month 18
GroupValue95% CI
HCV-infected Participants133.300± 50.2647
Month 24
GroupValue95% CI
HCV-infected Participants140.185± 97.2769
Month 30
GroupValue95% CI
HCV-infected Participants164.244± 276.9176
Month 36
GroupValue95% CI
HCV-infected Participants120.600
Mean FibroTest Score Over Time Secondary · From Day 1 up to 3 years post-treatment

A serum sample for FibroTest evaluation was collected at study visits for all participants, until the end of the study or initiation of re-treatment for HCV (if applicable). FibroTest uses six biomarkers to generate a score that correlates to the degree of liver damage in participants. Scores range from 0 to 1, with higher scores indicating more severe liver fibrosis.

Day 1
GroupValue95% CI
HCV-infected Participants0.354± 0.2341
Month 3
GroupValue95% CI
HCV-infected Participants0.363± 0.2383
Month 6
GroupValue95% CI
HCV-infected Participants0.353± 0.2311
Month 12
GroupValue95% CI
HCV-infected Participants0.405± 0.0919
Month 18
GroupValue95% CI
HCV-infected Participants0.272± 0.1281
Month 24
GroupValue95% CI
HCV-infected Participants0.321± 0.2156
Month 30
GroupValue95% CI
HCV-infected Participants0.261± 0.1667
Month 36
GroupValue95% CI
HCV-infected Participants0.190
Mean Aspartate Transaminase to Platelet Ratio Index (APRI) Over Time Secondary · From Day 1 up to 3 years post-treatment

A serum sample for aspartate transaminase evaluation and platelets were collected at study visits for all participants, until the end of the study or initiation of re-treatment for HCV (if applicable). The aspartate transaminase to platelet ratio index (APRI) is used to assess liver fibrosis in participants with chronic liver disease. Scores range from 0 to ≥ 2.0, with scores \< 0.5 predictive of no liver fibrosis; scores \>1.5 significant fibrosis; and scores \> 2.0 indicative of cirrhosis.

Day 1
GroupValue95% CI
HCV-infected Participants0.317± 0.2268
Month 3
GroupValue95% CI
HCV-infected Participants0.303± 0.2171
Month 6
GroupValue95% CI
HCV-infected Participants0.298± 0.2101
Month 12
GroupValue95% CI
HCV-infected Participants0.230
Month 18
GroupValue95% CI
HCV-infected Participants0.262± 0.0589
Month 24
GroupValue95% CI
HCV-infected Participants0.275± 0.1620
Month 30
GroupValue95% CI
HCV-infected Participants0.227± 0.1084
Month 36
GroupValue95% CI
HCV-infected Participants0.215± 0.1768
Mean FibroScan Scores Over Time Secondary · Up to 3 years post-treatment

The FibroScan test is used to assess liver fibrosis in participants with chronic liver disease. This was not performed as a study procedure, but any available results from source documents were summarized. For participants with Hepatitis C infection, a Fibroscan score of 2-7 kPa indicates no liver scarring or mild scarring; a score of 8 or 9 is associated with moderate liver scarring; 9-14 indicates severe liver scarring; and 14 or higher is indicative of advanced liver scarring, cirrhosis.

Day 1
GroupValue95% CI
HCV-infected Participants13.014± 12.6850
Month 3
GroupValue95% CI
HCV-infected Participants6.880± 4.6677
Month 6
GroupValue95% CI
HCV-infected Participants10.780± 5.9260
Month 12
GroupValue95% CI
HCV-infected Participants7.700± 4.2208
Month 18
GroupValue95% CI
HCV-infected Participants6.913± 2.8002
Month 24
GroupValue95% CI
HCV-infected Participants6.757± 3.1564
Month 30
GroupValue95% CI
HCV-infected Participants9.400

Sponsor's own description

This was a long-term follow-up study to evaluate the durability of sustained virologic response (SVR), persistence of direct-acting antiviral agent (DAA) resistance, and clinical outcomes for participants who received glecaprevir (ABT-493) and/or pibrentasvir (ABT-530) in prior AbbVie Phase 2 or 3 clinical studies for the treatment of chronic hepatitis C virus (HCV) infection.

Publications & conference data

No peer-reviewed publications indexed yet for this trial. Completed trials usually publish results within 12-18 months.

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Primary sources · FDA · ClinicalTrials.gov · EMA · SEC EDGAR · ChEMBL · Wikidata · full sourcing