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NCT00794781

A Phase 1, Multicenter, Open-Label, Dose-Escalation, Safety, Pharmacokinetic, and Pharmacodynamic Study of E6201 in Subjects With Advanced Solid Tumors

Completed Phase 1 Last updated 14 October 2022
What this trial tests

Phase 1 trial testing E6201 in Advanced Solid Tumors in 55 participants. Completed in 15 October 2015.

Timeline
22 June 2008
Primary endpoint
1 August 2011
15 October 2015

Quick facts

Lead sponsorEisai Inc.
PhasePhase 1
StatusCompleted
Study typeINTERVENTIONAL
Allocationna
Designsingle group
Maskingnone
Primary purposetreatment
Enrollment55
Start date22 June 2008
Primary completion1 August 2011
Estimated completion15 October 2015
Sites12 locations across United States

Drugs / interventions tested

Conditions studied

Sponsor

Eisai Inc. — full company profile →

Who can join

18 and older, any sex, with Advanced Solid Tumors. Patients with the condition only — healthy volunteers not accepted.

Sponsor's own description

The purpose of this study is to determine the maximum tolerated dose (MTD), safety, tolerability, pharmacokinetics, pharmacodynamics and preliminary anti-tumor activity of E6201 in subjects with advanced solid tumors.

Publications & conference data

8 peer-reviewed publications reference this trial (live from Europe PMC):

  1. Current Advances in the Treatment of BRAF-Mutant Melanoma.
    Patel H, Yacoub N, Mishra R, White A, et al · · 2020 · cited 133× · PMID 32092958 · DOI 10.3390/cancers12020482
  2. MEK1/2 Inhibitors: Molecular Activity and Resistance Mechanisms.
    Wu PK, Park JI. · · 2015 · cited 107× · PMID 26615130 · DOI 10.1053/j.seminoncol.2015.09.023
  3. Targeting the RAS oncogene.
    Takashima A, Faller DV. · · 2013 · cited 95× · PMID 23360111 · DOI 10.1517/14728222.2013.764990
  4. Overcoming resistance to BRAF inhibition in BRAF-mutated metastatic melanoma.
    Spagnolo F, Ghiorzo P, Queirolo P. · · 2014 · cited 92× · PMID 25344914 · DOI 10.18632/oncotarget.2602
  5. Sensitivity to the MEK inhibitor E6201 in melanoma cells is associated with mutant BRAF and wildtype PTEN status.
    Byron SA, Loch DC, Wellens CL, Wortmann A, et al · · 2012 · cited 35× · PMID 23039341 · DOI 10.1186/1476-4598-11-75
  6. Use of kinase inhibitors to correct ΔF508-CFTR function.
    Trzcinska-Daneluti AM, Nguyen L, Jiang C, Fladd C, et al · · 2012 · cited 28× · PMID 22700489 · DOI 10.1074/mcp.m111.016626
  7. Anti-tumor and anti-metastasis efficacy of E6201, a MEK1 inhibitor, in preclinical models of triple-negative breast cancer.
    Lee J, Lim B, Pearson T, Choi K, et al · · 2019 · cited 21× · PMID 30826934 · DOI 10.1007/s10549-019-05166-3
  8. E6201, an intravenous MEK1 inhibitor, achieves an exceptional response in BRAF V600E-mutated metastatic malignant melanoma with brain metastases.
    Babiker HM, Byron SA, Hendricks WPD, Elmquist WF, et al · · 2019 · cited 20× · PMID 30264293 · DOI 10.1007/s10637-018-0668-8

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Other trials of E6201

Trials testing the same drug.

Other recruiting trials for Advanced Solid Tumors

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Other Eisai Inc. trials

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Data sources for this page

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