Last reviewed · How we verify

RAAS, Inflammation, and Post-operative AF

NCT00141778 Phase 2/Phase 3 COMPLETED Results posted

Atrial fibrillation (AF) is the most prevalent, sustained type of irregular heartbeat and affects over 2 million Americans. Post-operative AF, which leads to significant morbidity and a prolonged hospital stay, complicates 20% to 40% of cardiopulmonary bypass (CPB) surgical procedures. While recent studies indicate that interruption of the renin-angiotensin-aldosterone system by either angiotensin-converting enzyme (ACE) inhibition or AT1 receptor antagonism decreases the incidence of AF following a heart attack or cardioversion (electric shock to the heart), its effect on the incidence of post-operative AF has not been throughly studied. Studies in both animals and humans suggest that inflammation-induced atrial remodeling plays an important role in the cause of AF. Recent studies also provide evidence that activation of the renin-angiotensin-aldosterone system induces inflammation, myocyte injury, proarrhythmic electrical remodeling, and fibrosis through aldosterone.

Details

Lead sponsorVanderbilt University
PhasePhase 2/Phase 3
StatusCOMPLETED
Enrolment455
Start date2005-04
Completion2010-08

Conditions

Interventions

Primary outcomes

Countries

United States