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Zenapax®, CellCept® and prednisolone

University of Oslo School of Pharmacy · FDA-approved active Small molecule

This combination suppresses the immune system to prevent organ rejection after transplantation by blocking T-cell activation and proliferation while reducing corticosteroid-induced inflammation.

This combination suppresses the immune system to prevent organ rejection after transplantation by blocking T-cell activation and proliferation while reducing corticosteroid-induced inflammation. Used for Prevention of acute organ rejection in renal transplant recipients, Prevention of acute organ rejection in other solid organ transplants.

At a glance

Generic nameZenapax®, CellCept® and prednisolone
SponsorUniversity of Oslo School of Pharmacy
Drug classImmunosuppressive combination therapy
TargetIL-2 receptor (daclizumab); inosine monophosphate dehydrogenase (mycophenolate mofetil); glucocorticoid receptor (prednisolone)
ModalitySmall molecule
Therapeutic areaImmunology / Transplantation
PhaseFDA-approved

Mechanism of action

Zenapax (daclizumab) is a monoclonal antibody that blocks the IL-2 receptor on T cells, preventing their activation. CellCept (mycophenolate mofetil) inhibits inosine monophosphate dehydrogenase, selectively suppressing T and B cell proliferation. Prednisolone is a corticosteroid that broadly suppresses immune responses. Together, this triple therapy provides multi-targeted immunosuppression for transplant rejection prevention.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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