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warfarine - low molecular heparin

University Hospital, Limoges · Phase 3 active Small molecule

Warfarin inhibits the synthesis of vitamin K-dependent clotting factors in the liver, preventing blood clot formation.

Warfarin inhibits the synthesis of vitamin K-dependent clotting factors in the liver, preventing blood clot formation. Used for Prevention of stroke and systemic embolism in patients with non-valvular atrial fibrillation, Treatment and prevention of deep vein thrombosis and pulmonary embolism.

At a glance

Generic namewarfarine - low molecular heparin
SponsorUniversity Hospital, Limoges
Drug classVitamin K antagonist
TargetVitamin K epoxide reductase
ModalitySmall molecule
Therapeutic areaCardiovascular
PhasePhase 3

Mechanism of action

Warfarin acts as an anticoagulant by inhibiting the gamma-carboxylation of vitamin K-dependent clotting factors, including prothrombin and factors VII, IX, and X. This inhibition prevents the formation of blood clots and reduces the risk of thromboembolic events. The exact mechanism of warfarin's action is complex and involves multiple pathways, including the inhibition of vitamin K epoxide reductase and the subsequent depletion of vitamin K-dependent clotting factors.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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