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Warfarin or coumadin

Ottawa Heart Institute Research Corporation · FDA-approved active Small molecule

Warfarin inhibits vitamin K-dependent clotting factors (II, VII, IX, X) by blocking the regeneration of reduced vitamin K, thereby reducing the formation of blood clots.

Warfarin inhibits vitamin K-dependent clotting factors (II, VII, IX, X) by blocking the regeneration of reduced vitamin K, thereby reducing the formation of blood clots. Used for Atrial fibrillation for stroke prevention, Venous thromboembolism (deep vein thrombosis and pulmonary embolism) treatment and prevention, Mechanical heart valve thromboprophylaxis.

At a glance

Generic nameWarfarin or coumadin
SponsorOttawa Heart Institute Research Corporation
Drug classVitamin K antagonist (oral anticoagulant)
TargetVitamin K epoxide reductase complex 1 (VKORC1)
ModalitySmall molecule
Therapeutic areaCardiovascular
PhaseFDA-approved

Mechanism of action

Warfarin acts as a vitamin K antagonist, interfering with the gamma-carboxylation of clotting factors II, VII, IX, and X in the liver. This prevents these factors from becoming fully activated and functional in the coagulation cascade. The result is a prolonged prothrombin time (PT/INR) and reduced thrombin generation, which decreases the risk of thrombotic events.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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