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Vyndaqel (Tafamidis Meglumine)

Foldrx Pharms · FDA-approved approved Small molecule Quality 60/100

Tafamidis selectively stabilizes TTR tetramer by binding at thyroxine sites, slowing monomer dissociation.

Tafamidis meglumine (Vyndaqel) is a transthyretin stabilizer indicated for treating cardiomyopathy of wild-type or hereditary transthyretin-mediated amyloidosis in adults. The drug demonstrates strong protein binding (>99%) with a half-life of approximately 49 hours and primarily hepatic clearance via glucuronidation. Key risk is BCRP inhibition, which may increase exposure to BCRP substrate drugs requiring monitoring and potential dose adjustment. Clinical efficacy is supported by reduction in cardiovascular mortality and cardiovascular-related hospitalization in ATTR-CM patients.

At a glance

Generic nameTafamidis Meglumine
SponsorFoldrx Pharms
Drug classTransthyretin stabilizer
TargetTransthyretin (TTR)
ModalitySmall molecule
Therapeutic areaNeuroscience
PhaseFDA-approved
First approval2019

Mechanism of action

Tafamidis is a selective stabilizer of transthyretin (TTR) that binds to TTR at the thyroxine binding sites. By binding at these sites, tafamidis stabilizes the TTR tetramer and slows its dissociation into monomers. This mechanism is significant because monomer dissociation is the rate-limiting step in the amyloidogenic process, the pathological cascade that leads to transthyretin-mediated amyloidosis.

Approved indications

Common side effects

Drug interactions

Patents

PatentExpiryType

Primary sources

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SourceUsed for
FDA labelMechanism, indications, dosing, boxed warnings, drug interactions
FDA Orange BookPatents + exclusivity

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