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VAL

Estetra · FDA-approved active Small molecule

VAL is a valganciclovir prodrug that is converted to ganciclovir in the body to inhibit viral DNA polymerase and prevent cytomegalovirus (CMV) replication.

VAL is a valganciclovir prodrug that is converted to ganciclovir in the body to inhibit viral DNA polymerase and prevent cytomegalovirus (CMV) replication. Used for Cytomegalovirus (CMV) retinitis in immunocompromised patients (including HIV/AIDS), CMV disease prevention in high-risk transplant recipients.

At a glance

Generic nameVAL
Also known asValproic acid, Depakote®
SponsorEstetra
Drug classNucleoside analog antiviral
TargetCytomegalovirus (CMV) DNA polymerase
ModalitySmall molecule
Therapeutic areaInfectious Disease / Virology
PhaseFDA-approved

Mechanism of action

Valganciclovir is an L-valyl ester prodrug of ganciclovir that achieves higher oral bioavailability than ganciclovir alone. Once absorbed, it is rapidly converted to ganciclovir by intestinal and hepatic esterases. Ganciclovir is then phosphorylated by viral thymidine kinase and cellular kinases to form the active triphosphate form, which inhibits viral DNA polymerase and prevents CMV DNA synthesis.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results