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US-licensed Prolia (Amgen)

Fresenius Kabi SwissBioSim GmbH · Phase 3 active Small molecule Under review Quality 0/100

US-licensed Prolia (Amgen) is a Denosumab Small molecule drug developed by Fresenius Kabi SwissBioSim GmbH. It is currently in Phase 3 development for Treatment of osteoporosis in postmenopausal women at high risk of fracture, treatment to increase bone mass in men with osteoporosis, treatment to increase bone mass in women with osteoporosis due to sustained systemic glucocorticoid therapy, treatment of bone loss associated with hormone ablation in men with prostate cancer, treatment of bone loss in women with breast cancer, treatment of giant cell tumor of bone, treatment of hypercalcemia of malignancy, treatment of multiple myeloma, treatment of bone metastases in men with prostate cancer, treatment of bone metastases in women with breast cancer. Also known as: Denosumab.

Prolia is a human monoclonal antibody that targets RANKL, a protein involved in bone resorption.

US-licensed Prolia (Amgen) is used to treat postmenopausal osteoporosis. It works by inhibiting interleukin-6, a protein involved in bone resorption.

Likelihood of approval
58.3% vs 58.3% industry baseline
If approved by FDA: likely 2028–2030
Steps remaining: NDA/BLA submission
Confidence: High
Why this estimate
  • Baseline phase 3 → approval rate +58.3pp
    Industry-wide phase 3 drugs reach approval ~58.3% of the time (BIO/Informa 2023 industry benchmark across all therapeutic areas).
Predicted approval windows by jurisdiction (conditional on FDA approval)
Regulator Country Likely year Lag vs FDA
FDA US 2028–2030
EMA EU 2029–2031 +0.7 yr
MHRA GB 2029–2031 +0.7 yr
Health Canada CA 2029–2032 +0.9 yr
TGA AU 2029–2032 +1.2 yr
PMDA JP 2029–2032 +1.5 yr
NMPA CN 2030–2033 +2.3 yr
MFDS KR 2029–2032 +1.4 yr
CDSCO IN 2029–2033 +1.8 yr
ANVISA BR 2030–2033 +2.3 yr

Hover any row for the lag rationale. Lag estimates are reduced when the drug has FDA Breakthrough or EMA PRIME designation (sponsors file globally in parallel).

Estimate based on the BIO/Informa industry phase transition rates plus per-drug modifiers for therapeutic area, sponsor type, FDA designations, mechanism, and trial design. Per-jurisdiction lags from Tufts CSDD international approval studies. Not investment, clinical or regulatory advice. Methodology: /methodology#likelihood.

At a glance

Generic nameUS-licensed Prolia (Amgen)
Also known asDenosumab
SponsorFresenius Kabi SwissBioSim GmbH
Drug classDenosumab
TargetRANKL
ModalitySmall molecule
Therapeutic areaOsteoporosis
PhasePhase 3

Mechanism of action

By binding to RANKL, Prolia inhibits the formation of osteoclasts, which are cells responsible for bone breakdown. This leads to an increase in bone mass and a decrease in the risk of fractures. Prolia is administered via subcutaneous injection and has a half-life of approximately 28 days.

Approved indications

Common side effects

Key clinical trials

Primary sources

Every claim on this page is sourced from regulatory or scientific primary sources. See our editorial policy for full methodology.

SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

Competitive intelligence

For the full competitive landscape — auto-detected comparators, recent regulatory actions across the set, upcoming PDUFA, patent timeline, sponsor landscape:

Frequently asked questions about US-licensed Prolia (Amgen)

What is US-licensed Prolia (Amgen)?

US-licensed Prolia (Amgen) is a Denosumab drug developed by Fresenius Kabi SwissBioSim GmbH, indicated for Treatment of osteoporosis in postmenopausal women at high risk of fracture, treatment to increase bone mass in men with osteoporosis, treatment to increase bone mass in women with osteoporosis due to sustained systemic glucocorticoid therapy, treatment of bone loss associated with hormone ablation in men with prostate cancer, treatment of bone loss in women with breast cancer, treatment of giant cell tumor of bone, treatment of hypercalcemia of malignancy, treatment of multiple myeloma, treatment of bone metastases in men with prostate cancer, treatment of bone metastases in women with breast cancer.

How does US-licensed Prolia (Amgen) work?

Prolia is a human monoclonal antibody that targets RANKL, a protein involved in bone resorption.

What is US-licensed Prolia (Amgen) used for?

US-licensed Prolia (Amgen) is indicated for Treatment of osteoporosis in postmenopausal women at high risk of fracture, treatment to increase bone mass in men with osteoporosis, treatment to increase bone mass in women with osteoporosis due to sustained systemic glucocorticoid therapy, treatment of bone loss associated with hormone ablation in men with prostate cancer, treatment of bone loss in women with breast cancer, treatment of giant cell tumor of bone, treatment of hypercalcemia of malignancy, treatment of multiple myeloma, treatment of bone metastases in men with prostate cancer, treatment of bone metastases in women with breast cancer.

Who makes US-licensed Prolia (Amgen)?

US-licensed Prolia (Amgen) is developed by Fresenius Kabi SwissBioSim GmbH (see full Fresenius Kabi SwissBioSim GmbH pipeline at /company/fresenius-kabi-swissbiosim-gmbh).

Is US-licensed Prolia (Amgen) also known as anything else?

US-licensed Prolia (Amgen) is also known as Denosumab.

What drug class is US-licensed Prolia (Amgen) in?

US-licensed Prolia (Amgen) belongs to the Denosumab class. See all Denosumab drugs at /class/denosumab.

What development phase is US-licensed Prolia (Amgen) in?

US-licensed Prolia (Amgen) is in Phase 3.

What are the side effects of US-licensed Prolia (Amgen)?

Common side effects of US-licensed Prolia (Amgen) include back pain, musculoskeletal pain, hypocalcemia, dizziness, nausea.

What does US-licensed Prolia (Amgen) target?

US-licensed Prolia (Amgen) targets RANKL and is a Denosumab.

Related

Primary sources · FDA · ClinicalTrials.gov · EMA · SEC EDGAR · ChEMBL · Wikidata · full sourcing