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Triamcinolone + Bevacizumab

Rubens Belfort Jr. · FDA-approved active Small molecule

Triamcinolone reduces inflammation via glucocorticoid receptor activation, while bevacizumab inhibits vascular endothelial growth factor (VEGF) to suppress neovascularization and reduce vascular permeability.

Triamcinolone reduces inflammation via glucocorticoid receptor activation, while bevacizumab inhibits vascular endothelial growth factor (VEGF) to suppress neovascularization and reduce vascular permeability. Used for Diabetic macular edema, Retinal vein occlusion with macular edema, Age-related macular degeneration.

At a glance

Generic nameTriamcinolone + Bevacizumab
Also known astriamcinolone + avastin
SponsorRubens Belfort Jr.
Drug classCorticosteroid + Monoclonal antibody (anti-VEGF)
TargetGlucocorticoid receptor; VEGF (vascular endothelial growth factor)
ModalitySmall molecule
Therapeutic areaOphthalmology
PhaseFDA-approved

Mechanism of action

This combination targets both inflammatory and angiogenic pathways in ocular disease. Triamcinolone is a corticosteroid that suppresses immune-mediated inflammation, while bevacizumab is a monoclonal antibody against VEGF that reduces pathological blood vessel formation and vascular leakage. Together, they address multiple pathogenic mechanisms in retinal and choroidal diseases.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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