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Tranexamic acid tablets

Carlos A Acosta-Olivo · Phase 3 active Small molecule

Tranexamic acid inhibits fibrinolysis by blocking plasminogen activation, thereby reducing blood clot breakdown and promoting hemostasis.

Tranexamic acid inhibits fibrinolysis by blocking plasminogen activation, thereby reducing blood clot breakdown and promoting hemostasis. Used for Reduction of blood loss in surgical procedures, Treatment of heavy menstrual bleeding, Prevention and treatment of bleeding in hemophilia patients.

At a glance

Generic nameTranexamic acid tablets
Also known asOral administration of tranexamic acid, Lysteda, XP12B, Kapron(R), Lysteda, Cyklokapron
SponsorCarlos A Acosta-Olivo
Drug classAntifibrinolytic agent
TargetPlasminogen / Plasmin
ModalitySmall molecule
Therapeutic areaHematology / Hemostasis
PhasePhase 3

Mechanism of action

Tranexamic acid is a competitive inhibitor of plasminogen and plasmin, the key enzymes responsible for breaking down fibrin clots. By preventing the conversion of plasminogen to plasmin, it stabilizes formed clots and reduces excessive bleeding. This antifibrinolytic mechanism makes it useful in conditions characterized by excessive or prolonged bleeding.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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