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Tranexamic Acid / Sodium chloride

University Hospital, Bordeaux · FDA-approved active Small molecule

Tranexamic acid inhibits fibrinolysis by blocking plasminogen activation, thereby reducing bleeding and promoting clot stability.

Tranexamic acid inhibits fibrinolysis by blocking plasminogen activation, thereby reducing bleeding and promoting clot stability. Used for Excessive bleeding or hemorrhage (perioperative, trauma, or menorrhagia), Prevention of blood loss in surgical procedures.

At a glance

Generic nameTranexamic Acid / Sodium chloride
SponsorUniversity Hospital, Bordeaux
Drug classAntifibrinolytic agent
TargetPlasminogen / Plasmin
ModalitySmall molecule
Therapeutic areaHematology / Hemostasis
PhaseFDA-approved

Mechanism of action

Tranexamic acid is a competitive inhibitor of plasminogen and plasmin, enzymes responsible for breaking down fibrin clots. By preventing fibrin degradation, it stabilizes blood clots and reduces excessive bleeding. The sodium chloride component is a physiological saline carrier for intravenous administration.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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