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Ticagrelor or Prasugrel

St. Antonius Hospital · FDA-approved active Small molecule ✓ Verified May 2026

Ticagrelor or Prasugrel is a P2Y12 receptor antagonist Small molecule drug developed by St. Antonius Hospital. It is currently FDA-approved for Acute coronary syndrome (ACS) including unstable angina, NSTEMI, and STEMI, Secondary prevention after percutaneous coronary intervention (PCI), Reduction of thrombotic cardiovascular events in patients with established coronary artery disease. Also known as: Brilinta, Prasuvas.

Both ticagrelor and prasugrel are P2Y12 receptor antagonists that inhibit platelet aggregation by blocking ADP-mediated platelet activation.

Prasugrel is a small molecule used to treat conditions such as myocardial infarction, atrial fibrillation, and platelet dysfunction. It is used in conjunction with other medications, including edoxaban and vitamin K antagonists, in patients undergoing percutaneous coronary intervention.

At a glance

Generic nameTicagrelor or Prasugrel
Also known asBrilinta, Prasuvas
SponsorSt. Antonius Hospital
Drug classP2Y12 receptor antagonist
TargetP2Y12 receptor
ModalitySmall molecule
Therapeutic areaCardiovascular
PhaseFDA-approved

Mechanism of action

These drugs bind irreversibly (prasugrel) or reversibly (ticagrelor) to the P2Y12 adenosine diphosphate receptor on platelet surfaces, preventing platelet clumping and thrombus formation. They are used as antiplatelet agents in acute coronary syndromes and after percutaneous coronary intervention to reduce cardiovascular events.

Approved indications

Common side effects

Key clinical trials

Primary sources

Every claim on this page is sourced from regulatory or scientific primary sources. See our editorial policy for full methodology.

SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

Competitive intelligence

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Frequently asked questions about Ticagrelor or Prasugrel

What is Ticagrelor or Prasugrel?

Ticagrelor or Prasugrel is a P2Y12 receptor antagonist drug developed by St. Antonius Hospital, indicated for Acute coronary syndrome (ACS) including unstable angina, NSTEMI, and STEMI, Secondary prevention after percutaneous coronary intervention (PCI), Reduction of thrombotic cardiovascular events in patients with established coronary artery disease.

How does Ticagrelor or Prasugrel work?

Both ticagrelor and prasugrel are P2Y12 receptor antagonists that inhibit platelet aggregation by blocking ADP-mediated platelet activation.

What is Ticagrelor or Prasugrel used for?

Ticagrelor or Prasugrel is indicated for Acute coronary syndrome (ACS) including unstable angina, NSTEMI, and STEMI, Secondary prevention after percutaneous coronary intervention (PCI), Reduction of thrombotic cardiovascular events in patients with established coronary artery disease.

Who makes Ticagrelor or Prasugrel?

Ticagrelor or Prasugrel is developed and marketed by St. Antonius Hospital (see full St. Antonius Hospital pipeline at /company/st-antonius-hospital).

Is Ticagrelor or Prasugrel also known as anything else?

Ticagrelor or Prasugrel is also known as Brilinta, Prasuvas.

What drug class is Ticagrelor or Prasugrel in?

Ticagrelor or Prasugrel belongs to the P2Y12 receptor antagonist class. See all P2Y12 receptor antagonist drugs at /class/p2y12-receptor-antagonist.

What development phase is Ticagrelor or Prasugrel in?

Ticagrelor or Prasugrel is FDA-approved (marketed).

What are the side effects of Ticagrelor or Prasugrel?

Common side effects of Ticagrelor or Prasugrel include Bleeding (major and minor), Dyspnea (ticagrelor), Bradycardia (ticagrelor), Thrombotic thrombocytopenic purpura (prasugrel).

What does Ticagrelor or Prasugrel target?

Ticagrelor or Prasugrel targets P2Y12 receptor and is a P2Y12 receptor antagonist.

Related

Primary sources · FDA · ClinicalTrials.gov · EMA · SEC EDGAR · ChEMBL · Wikidata · full sourcing