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Ticagrelor + ASA + Bivalirudin

Juan J Badimon · FDA-approved active Small molecule

This combination uses ticagrelor and aspirin to inhibit platelet aggregation via P2Y12 and COX-1 pathways, while bivalirudin provides direct thrombin inhibition to prevent thrombotic events.

This combination uses ticagrelor and aspirin to inhibit platelet aggregation via P2Y12 and COX-1 pathways, while bivalirudin provides direct thrombin inhibition to prevent thrombotic events. Used for Acute coronary syndrome with percutaneous coronary intervention, Prevention of stent thrombosis and recurrent ischemic events.

At a glance

Generic nameTicagrelor + ASA + Bivalirudin
Also known asBrilinta (ticagrelor, Aspirin (ASA), Angiomax (bivalirudin)
SponsorJuan J Badimon
Drug classAntiplatelet agent + anticoagulant combination
TargetP2Y12 receptor, COX-1, thrombin
ModalitySmall molecule
Therapeutic areaCardiovascular
PhaseFDA-approved

Mechanism of action

Ticagrelor is a P2Y12 receptor antagonist that blocks ADP-mediated platelet activation, while aspirin irreversibly inhibits cyclooxygenase-1 to reduce thromboxane A2 production. Bivalirudin is a direct thrombin inhibitor that prevents fibrin formation and thrombus propagation. Together, this triple antithrombotic regimen targets multiple pathways of coagulation and platelet function to reduce ischemic cardiovascular events.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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