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Micardis (TELMISARTAN)

Boehringer Ingelheim · FDA-approved approved Small molecule Verified Quality 80/100

Micardis works by blocking the action of angiotensin II, a hormone that constricts blood vessels and increases blood pressure.

Telmisartan (Micardis), marketed by Boehringer Ingelheim, is an angiotensin II receptor blocker primarily indicated for hypertension, with a key composition patent expiring in 2028. Its key strength lies in its well-established market presence and ongoing patent protection, which differentiates it from off-patent competitors like eprosartan, valsartan, irbesartan, and candesartan cilexetil. The primary risk is the increasing competition from generics, particularly from drugs like valsartan and irbesartan, which have numerous generic formulations already available.

At a glance

Generic nameTELMISARTAN
SponsorBoehringer Ingelheim
Drug classAngiotensin 2 Receptor Blocker [EPC]
TargetType-2 angiotensin II receptor
ModalitySmall molecule
Therapeutic areaMetabolic
PhaseFDA-approved
First approval1998

Mechanism of action

Angiotensin II is formed from angiotensin in reaction catalyzed by angiotensin-converting enzyme (ACE, kininase II). Angiotensin II is the principal pressor agent of the reninangiotensin system, with effects that include vasoconstriction, stimulation of synthesis and release of aldosterone, cardiac stimulation, and renal reabsorption of sodium. Telmisartan blocks the vasoconstrictor and aldosterone-secreting effects of angiotensin II by selectively blocking the binding of angiotensin II to the AT1 receptor in many tissues, such as vascular smooth muscle and the adrenal gland. Its action is therefore independent of the pathways for angiotensin II synthesis.There is also an AT2 receptor found in many tissues, but AT2 is not known to be associated with cardiovascular homeostasis. Telmisartan has much greater affinity (>3,000 fold) for the AT1 receptor than for the AT2 receptor.Blockade of the renin-angiotensin system with ACE inhibitors, which inhibit the biosynt

Approved indications

Boxed warnings

Common side effects

Drug interactions

Key clinical trials

Primary sources

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SourceUsed for
FDA labelMechanism, indications, dosing, boxed warnings, drug interactions
ClinicalTrials.govTrial enrolment, design, endpoints, results

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