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Tacrolimus (Arm B)

Weill Medical College of Cornell University · FDA-approved active Small molecule

Tacrolimus inhibits calcineurin phosphatase, blocking T-cell activation and proliferation by preventing nuclear translocation of NFAT transcription factors.

Tacrolimus inhibits calcineurin phosphatase, blocking T-cell activation and proliferation by preventing nuclear translocation of NFAT transcription factors. Used for Organ transplant rejection prophylaxis (kidney, heart, liver, pancreas), Atopic dermatitis, Autoimmune conditions (off-label use in various settings).

At a glance

Generic nameTacrolimus (Arm B)
Also known asPrograf
SponsorWeill Medical College of Cornell University
Drug classCalcineurin inhibitor
TargetCalcineurin (via FKBP12 binding)
ModalitySmall molecule
Therapeutic areaImmunology
PhaseFDA-approved

Mechanism of action

Tacrolimus binds to the immunophilin FKBP12, and this complex inhibits calcineurin, a serine/threonine phosphatase essential for dephosphorylating NFAT. This prevents NFAT from entering the nucleus and activating genes required for T-cell cytokine production and proliferation. The result is potent immunosuppression, making it effective for preventing organ rejection and treating autoimmune conditions.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results