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Sunitinib + Capecitabine

Pfizer · Phase 3 active Small molecule

Sunitinib inhibits multiple receptor tyrosine kinases, including VEGFR, PDGFR, and c-KIT, while Capecitabine is a prodrug that is converted into 5-fluorouracil, a thymidylate synthase inhibitor.

Sunitinib inhibits multiple receptor tyrosine kinases, including VEGFR, PDGFR, and c-KIT, while Capecitabine is a prodrug that is converted into 5-fluorouracil, a thymidylate synthase inhibitor. Used for Renal cell carcinoma, Gastrointestinal stromal tumor.

At a glance

Generic nameSunitinib + Capecitabine
SponsorPfizer
Drug classtyrosine kinase inhibitor, antimetabolite
TargetVEGFR, PDGFR, c-KIT, thymidylate synthase
ModalitySmall molecule
Therapeutic areaOncology
PhasePhase 3

Mechanism of action

Sunitinib's inhibition of VEGFR and PDGFR leads to anti-angiogenic effects, while its inhibition of c-KIT can affect tumor growth. Capecitabine's conversion to 5-fluorouracil results in the inhibition of thymidylate synthase, leading to DNA damage and cell death in rapidly dividing cancer cells.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results