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Solifenacin + Mirabegron

University of Aarhus · FDA-approved active Small molecule

Solifenacin and mirabegron work synergistically to reduce overactive bladder symptoms by blocking muscarinic receptors (solifenacin) and activating beta-3 adrenergic receptors (mirabegron) to decrease detrusor muscle contractions and increase bladder capacity.

Solifenacin and mirabegron work synergistically to reduce overactive bladder symptoms by blocking muscarinic receptors (solifenacin) and activating beta-3 adrenergic receptors (mirabegron) to decrease detrusor muscle contractions and increase bladder capacity. Used for Overactive bladder with symptoms of urgency, frequency, and urgency incontinence.

At a glance

Generic nameSolifenacin + Mirabegron
SponsorUniversity of Aarhus
Drug classAntimuscarinic + Beta-3 adrenergic agonist combination
TargetM3 muscarinic receptor (solifenacin) and beta-3 adrenergic receptor (mirabegron)
ModalitySmall molecule
Therapeutic areaUrology / Neurology
PhaseFDA-approved

Mechanism of action

Solifenacin is an antimuscarinic agent that blocks M3 receptors on bladder smooth muscle, reducing involuntary contractions. Mirabegron is a beta-3 adrenergic agonist that relaxes the detrusor muscle during the storage phase of the bladder. Together, they provide dual mechanism action targeting both parasympathetic and sympathetic pathways to improve bladder storage capacity and reduce urgency and frequency.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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