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Sodium Valproate treatment

University of Pittsburgh · FDA-approved active Small molecule

Sodium valproate inhibits histone deacetylase (HDAC) and enhances gamma-aminobutyric acid (GABA) neurotransmission in the central nervous system.

Sodium valproate inhibits histone deacetylase (HDAC) and enhances gamma-aminobutyric acid (GABA) neurotransmission in the central nervous system. Used for Epilepsy and seizure disorders, Bipolar disorder, Migraine prophylaxis.

At a glance

Generic nameSodium Valproate treatment
Also known asDepakote
SponsorUniversity of Pittsburgh
Drug classHistone deacetylase inhibitor; anticonvulsant
TargetHistone deacetylase (HDAC); GABA metabolism
ModalitySmall molecule
Therapeutic areaNeurology; Oncology
PhaseFDA-approved

Mechanism of action

Sodium valproate increases GABA levels by inhibiting GABA catabolism and enhancing GABA synthesis, leading to increased inhibitory neurotransmission. Additionally, it acts as a histone deacetylase inhibitor, which affects gene expression and has been investigated for epigenetic effects in various conditions including cancer and neurological disorders.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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