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Sodium-glucose cotransporter 2 inhibitor

Queen's University, Belfast · FDA-approved active Small molecule

SGLT2 inhibitors block the sodium-glucose cotransporter 2 in the kidney, preventing reabsorption of glucose and increasing urinary glucose excretion.

SGLT2 inhibitors block the sodium-glucose cotransporter 2 in the kidney, preventing reabsorption of glucose and increasing urinary glucose excretion. Used for Type 2 diabetes mellitus, Heart failure with reduced ejection fraction, Chronic kidney disease.

At a glance

Generic nameSodium-glucose cotransporter 2 inhibitor
Also known asSGLT2 inhibitors, dapagliflozin, canagliflozin, empagliflozin
SponsorQueen's University, Belfast
Drug classSGLT2 inhibitor
TargetSGLT2 (sodium-glucose cotransporter 2)
ModalitySmall molecule
Therapeutic areaDiabetes, Cardiovascular, Nephrology
PhaseFDA-approved

Mechanism of action

SGLT2 is a transporter protein in the proximal tubule of the nephron responsible for reabsorbing filtered glucose back into the bloodstream. By inhibiting this transporter, SGLT2 inhibitors reduce blood glucose levels by promoting urinary glucose loss, independent of insulin secretion. This mechanism also produces osmotic diuresis and has cardioprotective and renoprotective effects beyond glucose lowering.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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