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SGLT2i

University of Chicago · FDA-approved active Small molecule

SGLT2 inhibitors block sodium-glucose cotransporter 2 in the kidney, reducing glucose reabsorption and increasing urinary glucose excretion.

SGLT2 inhibitors block sodium-glucose cotransporter 2 in the kidney, reducing glucose reabsorption and increasing urinary glucose excretion. Used for Type 2 diabetes mellitus, Heart failure with reduced ejection fraction, Chronic kidney disease.

At a glance

Generic nameSGLT2i
SponsorUniversity of Chicago
Drug classSGLT2 inhibitor
TargetSGLT2 (sodium-glucose cotransporter 2)
ModalitySmall molecule
Therapeutic areaDiabetes
PhaseFDA-approved

Mechanism of action

SGLT2 inhibitors work by inhibiting the SGLT2 transporter protein located in the proximal tubule of the kidney. This prevents the reabsorption of filtered glucose, leading to increased urinary glucose excretion (glucosuria) and lower blood glucose levels. Beyond glycemic control, SGLT2 inhibitors provide cardiovascular and renal protective effects through mechanisms including osmotic diuresis, improved insulin sensitivity, and reduced intraglomerular pressure.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results