Last reviewed · How we verify

SGLT-2 inhibitors

Second Affiliated Hospital, School of Medicine, Zhejiang University · FDA-approved active Small molecule

SGLT-2 inhibitors block sodium-glucose cotransporter 2 in the kidney to reduce glucose reabsorption and increase urinary glucose excretion.

SGLT-2 inhibitors block sodium-glucose cotransporter 2 in the kidney to reduce glucose reabsorption and increase urinary glucose excretion. Used for Type 2 diabetes mellitus, Heart failure with reduced ejection fraction, Chronic kidney disease.

At a glance

Generic nameSGLT-2 inhibitors
SponsorSecond Affiliated Hospital, School of Medicine, Zhejiang University
Drug classSGLT-2 inhibitor
TargetSGLT-2 (sodium-glucose cotransporter 2)
ModalitySmall molecule
Therapeutic areaDiabetes, Cardiovascular, Nephrology
PhaseFDA-approved

Mechanism of action

SGLT-2 inhibitors work by inhibiting the sodium-glucose cotransporter 2 (SGLT-2) protein in the proximal tubule of the kidney. This transporter normally reabsorbs filtered glucose back into the bloodstream. By blocking this transporter, these drugs allow glucose to be excreted in the urine, thereby lowering blood glucose levels independently of insulin secretion. Beyond glycemic control, SGLT-2 inhibitors provide cardiovascular and renal protective effects through mechanisms including osmotic diuresis, reduced intraglomerular pressure, and improved metabolic parameters.

Approved indications

Common side effects

Key clinical trials

Primary sources

Every claim on this page is sourced from regulatory or scientific primary sources. See our editorial policy for full methodology.

SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

Competitive intelligence

For the full competitive landscape — auto-detected comparators, recent regulatory actions across the set, upcoming PDUFA, patent timeline, sponsor landscape: