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Romosozumab followed by Denosumab

National Taiwan University Hospital · FDA-approved active Small molecule

Romosozumab stimulates bone formation via Wnt signaling inhibition, followed by Denosumab which blocks osteoclast-mediated bone resorption, providing sequential anabolic then anti-catabolic therapy for osteoporosis.

Romosozumab stimulates bone formation via Wnt signaling inhibition, followed by Denosumab which blocks osteoclast-mediated bone resorption, providing sequential anabolic then anti-catabolic therapy for osteoporosis. Used for Osteoporosis in postmenopausal women at high risk of fracture.

At a glance

Generic nameRomosozumab followed by Denosumab
Also known asSequential therapy group
SponsorNational Taiwan University Hospital
Drug classSclerostin inhibitor followed by RANKL inhibitor
TargetSclerostin (romosozumab); RANKL (Denosumab)
ModalitySmall molecule
Therapeutic areaBone metabolism / Osteoporosis
PhaseFDA-approved

Mechanism of action

Romosozumab is a sclerostin inhibitor that promotes osteoblast activity and new bone formation while transiently reducing bone resorption. Denosumab is a RANKL inhibitor that potently suppresses osteoclast differentiation and function, reducing bone turnover. This sequential approach leverages the anabolic phase of romosozumab followed by sustained anti-resorptive effects of denosumab to maximize bone mineral density gains and fracture risk reduction.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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