Last reviewed · How we verify

Rivaroxaban or Marcumar

RWTH Aachen University · FDA-approved active Small molecule

Rivaroxaban is a direct Factor Xa inhibitor that prevents blood clot formation by blocking a key enzyme in the coagulation cascade, while Marcumar (phenprocoumon) is a vitamin K antagonist that inhibits clotting factor synthesis.

Rivaroxaban is a direct Factor Xa inhibitor that prevents blood clot formation by blocking a key enzyme in the coagulation cascade, while Marcumar (phenprocoumon) is a vitamin K antagonist that inhibits clotting factor synthesis. Used for Atrial fibrillation for stroke prevention, Deep vein thrombosis (DVT) treatment and prevention, Pulmonary embolism (PE) treatment and prevention.

At a glance

Generic nameRivaroxaban or Marcumar
Also known asXarelto; Marcumar
SponsorRWTH Aachen University
Drug classAnticoagulant (Rivaroxaban: direct Factor Xa inhibitor; Marcumar: vitamin K antagonist)
TargetFactor Xa (Rivaroxaban); Vitamin K epoxide reductase complex (Marcumar)
ModalitySmall molecule
Therapeutic areaCardiovascular
PhaseFDA-approved

Mechanism of action

Rivaroxaban selectively inhibits Factor Xa, a serine protease essential for thrombin generation and clot formation, providing rapid anticoagulation. Marcumar works through a different mechanism by antagonizing vitamin K-dependent clotting factors (II, VII, IX, X), requiring several days to achieve full effect. These are distinct anticoagulant classes with different pharmacokinetics and monitoring requirements.

Approved indications

Common side effects

Key clinical trials

Primary sources

Every claim on this page is sourced from regulatory or scientific primary sources. See our editorial policy for full methodology.

SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

Competitive intelligence

For the full competitive landscape — auto-detected comparators, recent regulatory actions across the set, upcoming PDUFA, patent timeline, sponsor landscape: