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Rapid-tapering glucocorticoids + Inebilizumab

Tianjin Medical University General Hospital · Phase 3 active Small molecule

Inebilizumab, a monoclonal antibody targeting plasmacytoid dendritic cells (pDCs), reduces pathogenic B cell and autoantibody production, combined with rapid-tapering glucocorticoids to suppress acute inflammation in autoimmune disease.

Inebilizumab, a monoclonal antibody targeting plasmacytoid dendritic cells (pDCs), reduces pathogenic B cell and autoantibody production, combined with rapid-tapering glucocorticoids to suppress acute inflammation in autoimmune disease. Used for Anti-aquaporin-4 (AQP4) antibody-positive neuromyelitis optica spectrum disorder (NMOSD).

At a glance

Generic nameRapid-tapering glucocorticoids + Inebilizumab
SponsorTianjin Medical University General Hospital
Drug classMonoclonal antibody + Glucocorticoid combination
TargetBDCA-2 (on pDCs) + Glucocorticoid receptor
ModalitySmall molecule
Therapeutic areaImmunology / Autoimmune disease
PhasePhase 3

Mechanism of action

Inebilizumab binds to BDCA-2 on plasmacytoid dendritic cells, depleting these cells and reducing their production of type I interferons and support for autoreactive B cells. Glucocorticoids provide rapid anti-inflammatory and immunosuppressive effects during the acute phase. Together, this combination targets both innate immune activation and adaptive autoimmune responses.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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