Last reviewed · How we verify

PUVA (8-MOP + UVA)

Madrilenian Group of Cutaneous Lymphomas · FDA-approved active Small molecule

PUVA therapy uses 8-methoxypsoralen (8-MOP) as a photosensitizing agent combined with UVA radiation to induce apoptosis in malignant T cells and suppress immune-mediated skin inflammation.

PUVA combines 8-methoxypsoralen (8-MOP), a photosensitizing agent, with UVA radiation to create DNA cross-links in rapidly dividing cells, inducing apoptosis in malignant lymphocytes. Used for Cutaneous T-cell lymphoma (mycosis fungoides), Cutaneous B-cell lymphoma, Severe psoriasis.

At a glance

Generic namePUVA (8-MOP + UVA)
SponsorMadrilenian Group of Cutaneous Lymphomas
Drug classPhotochemotherapy agent
TargetDNA (non-specific cross-linking)
ModalitySmall molecule
Therapeutic areaOncology
PhaseFDA-approved

Mechanism of action

8-MOP is an oral psoralen that intercalates into DNA when activated by UVA light (320-400 nm), creating thymine dimers and generating reactive oxygen species that trigger apoptosis in cutaneous lymphocytes. This mechanism is particularly effective against malignant T cells in cutaneous lymphomas while also providing immunosuppressive effects that benefit inflammatory skin conditions. The combination of photochemical DNA damage and immune modulation makes PUVA effective for both neoplastic and inflammatory cutaneous disorders.

Approved indications

Common side effects

Key clinical trials

Primary sources

Every claim on this page is sourced from regulatory or scientific primary sources. See our editorial policy for full methodology.

SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

Competitive intelligence

For the full competitive landscape — auto-detected comparators, recent regulatory actions across the set, upcoming PDUFA, patent timeline, sponsor landscape: