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prostaglandins E2

Tel-Aviv Sourasky Medical Center · Phase 3 active Small molecule

Prostaglandin E2 (PGE2) binds to EP prostanoid receptors on cell surfaces to modulate inflammation, immune responses, and tissue remodeling.

Prostaglandin E2 (PGE2) binds to EP prostanoid receptors on cell surfaces to modulate inflammation, immune responses, and tissue remodeling. Used for Graft-versus-host disease (GVHD) or related immune/inflammatory conditions (Phase 3 status suggests clinical development in this area).

At a glance

Generic nameprostaglandins E2
Also known asdinoprostone slow release pessary
SponsorTel-Aviv Sourasky Medical Center
Drug classProstanoid agonist
TargetEP prostanoid receptors (EP1, EP2, EP3, EP4)
ModalitySmall molecule
Therapeutic areaImmunology / Regenerative Medicine
PhasePhase 3

Mechanism of action

PGE2 is an endogenous eicosanoid that acts through four G-protein coupled EP receptors (EP1–EP4), triggering diverse downstream signaling pathways. Depending on the receptor subtype and tissue context, PGE2 can suppress or enhance inflammatory responses, promote vasodilation, inhibit platelet aggregation, and modulate bone metabolism and immune cell differentiation. Exogenous PGE2 administration aims to harness these pleiotropic effects therapeutically.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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