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Prograf (Tacrolimus)

East Carolina University · FDA-approved active Small molecule

Tacrolimus inhibits calcineurin phosphatase, blocking T-cell activation and proliferation to suppress the immune system.

Tacrolimus inhibits calcineurin phosphatase, blocking T-cell activation and proliferation to suppress the immune system. Used for Prophylaxis of organ rejection in allogeneic kidney, heart, and liver transplant recipients, Atopic dermatitis (topical formulation), Autoimmune conditions including rheumatoid arthritis and lupus nephritis.

At a glance

Generic namePrograf (Tacrolimus)
Also known asTacrolimus/Prograf®/FK506, FK506, tacrolimus
SponsorEast Carolina University
Drug classCalcineurin inhibitor
TargetCalcineurin (via FKBP12 binding)
ModalitySmall molecule
Therapeutic areaImmunology
PhaseFDA-approved

Mechanism of action

Tacrolimus binds to the immunophilin FKBP12, and this complex inhibits calcineurin, a key phosphatase required for dephosphorylation and nuclear translocation of NFAT (nuclear factor of activated T cells). This prevents T-cell receptor signaling and IL-2 production, thereby suppressing T-cell mediated immune responses. It is more potent than cyclosporine and is widely used in transplantation and autoimmune conditions.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results