Last reviewed · How we verify

prasugrel or ticagrelor

University of Florida · FDA-approved active Small molecule

Both prasugrel and ticagrelor are P2Y12 platelet adenosine diphosphate receptor antagonists that inhibit platelet aggregation and reduce thrombotic events.

Both prasugrel and ticagrelor are P2Y12 platelet adenosine diphosphate receptor antagonists that inhibit platelet aggregation and reduce thrombotic events. Used for Acute coronary syndrome (ACS) including ST-elevation myocardial infarction (STEMI) and non-ST-elevation acute coronary syndrome (NSTE-ACS), Prevention of stent thrombosis in patients undergoing percutaneous coronary intervention (PCI).

At a glance

Generic nameprasugrel or ticagrelor
Also known asEfient, Brilique
SponsorUniversity of Florida
Drug classP2Y12 receptor antagonist
TargetP2Y12 receptor
ModalitySmall molecule
Therapeutic areaCardiovascular
PhaseFDA-approved

Mechanism of action

Prasugrel and ticagrelor are dual antiplatelet agents that irreversibly (prasugrel) or reversibly (ticagrelor) block the P2Y12 receptor on platelets, preventing ADP-induced platelet activation and aggregation. This mechanism reduces the formation of pathological blood clots in coronary arteries and stents, lowering the risk of myocardial infarction and stent thrombosis in acute coronary syndrome patients.

Approved indications

Common side effects

Key clinical trials

Primary sources

Every claim on this page is sourced from regulatory or scientific primary sources. See our editorial policy for full methodology.

SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

Competitive intelligence

For the full competitive landscape — auto-detected comparators, recent regulatory actions across the set, upcoming PDUFA, patent timeline, sponsor landscape: