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Plain aspirin

Royal College of Surgeons, Ireland · FDA-approved active Small molecule

Aspirin irreversibly inhibits cyclooxygenase (COX) enzymes, reducing the production of prostaglandins and thromboxane to decrease inflammation, pain, and blood clotting.

Aspirin irreversibly inhibits cyclooxygenase (COX) enzymes, reducing the production of prostaglandins and thromboxane to decrease inflammation, pain, and blood clotting. Used for Acute myocardial infarction prevention and secondary prevention, Ischemic stroke prevention, Transient ischemic attack (TIA) prevention.

At a glance

Generic namePlain aspirin
Also known asLow dose of plain aspirin
SponsorRoyal College of Surgeons, Ireland
Drug classNonsteroidal anti-inflammatory drug (NSAID); antiplatelet agent
TargetCyclooxygenase-1 (COX-1); Cyclooxygenase-2 (COX-2)
ModalitySmall molecule
Therapeutic areaCardiovascular; Pain management; Inflammation
PhaseFDA-approved

Mechanism of action

Aspirin acetylates serine residues on COX-1 and COX-2 enzymes, permanently blocking their catalytic activity. This prevents the synthesis of prostaglandins (which mediate inflammation and pain) and thromboxane A2 (which promotes platelet aggregation). The antiplatelet effect is particularly important at low doses, where COX-1 inhibition in platelets predominates, making aspirin useful for cardiovascular protection.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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