Last reviewed · How we verify

Placebo to glipizide

Merck Sharp & Dohme LLC · Phase 3 active Small molecule

Glipizide stimulates insulin secretion from pancreatic beta cells by blocking ATP-sensitive potassium channels, thereby lowering blood glucose in type 2 diabetes.

Glipizide stimulates insulin secretion from pancreatic beta cells by blocking ATP-sensitive potassium channels, thereby lowering blood glucose in type 2 diabetes. Used for Type 2 diabetes mellitus.

At a glance

Generic namePlacebo to glipizide
SponsorMerck Sharp & Dohme LLC
Drug classSulfonylurea
TargetATP-sensitive potassium channel (KATP channel)
ModalitySmall molecule
Therapeutic areaDiabetes
PhasePhase 3

Mechanism of action

Glipizide is a second-generation sulfonylurea that binds to and closes ATP-sensitive potassium channels on the surface of pancreatic beta cells. This closure depolarizes the cell membrane, triggering calcium influx and subsequent insulin exocytosis. The increased circulating insulin reduces blood glucose levels in patients with type 2 diabetes mellitus.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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