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Placebo & Aspirin

Baim Institute for Clinical Research · FDA-approved active Small molecule

Aspirin inhibits platelet aggregation by irreversibly blocking cyclooxygenase (COX) enzymes, reducing thromboxane A2 production and preventing clot formation.

Aspirin inhibits platelet aggregation by irreversibly blocking cyclooxygenase (COX) enzymes, reducing thromboxane A2 production and preventing clot formation. Used for Cardiovascular event prevention (secondary prevention post-myocardial infarction or stroke), Acute coronary syndrome.

At a glance

Generic namePlacebo & Aspirin
SponsorBaim Institute for Clinical Research
Drug classAntiplatelet agent / NSAID
TargetCyclooxygenase-1 (COX-1), Cyclooxygenase-2 (COX-2)
ModalitySmall molecule
Therapeutic areaCardiovascular
PhaseFDA-approved

Mechanism of action

Aspirin acetylates serine residues on COX-1 and COX-2 enzymes, permanently inactivating them and preventing the synthesis of thromboxane A2 in platelets. This reduces platelet activation and aggregation, thereby decreasing the risk of thrombotic events. The placebo component serves as a control in clinical trials to assess the true efficacy of aspirin.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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