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PF-06462700

Pfizer · Phase 3 active Biologic

PF-06462700 is a PD-1 inhibitor Biologic drug developed by Pfizer. It is currently in Phase 3 development for Non-small cell lung cancer, PD-L1 positive, Head and neck squamous cell carcinoma. Also known as: Brand name in the US: ATGAM.

PF-06462700 is a small molecule that targets the PD-1 receptor.

PF-06462700 is a small molecule that targets the PD-1 receptor. Used for Non-small cell lung cancer, PD-L1 positive, Head and neck squamous cell carcinoma.

Likelihood of approval
64.3% vs 58.3% industry baseline
If approved by FDA: likely 2028–2030
Steps remaining: NDA/BLA submission
Confidence: High
Why this estimate
  • Baseline phase 3 → approval rate +58.3pp
    Industry-wide phase 3 drugs reach approval ~58.3% of the time (BIO/Informa 2023 industry benchmark across all therapeutic areas).
  • Oncology Phase 3 boost +3.0pp
    Oncology Phase 3 trials have higher approval rates (~61%) than the cross-industry average due to clearer endpoints and FDA oncology pathway.
  • Big-pharma sponsor +3.0pp
    Pfizer is a top-20 pharma sponsor — historical approval rates run ~3pp above average due to scale, regulatory experience, and trial-design quality.
Predicted approval windows by jurisdiction (conditional on FDA approval)
Regulator Country Likely year Lag vs FDA
FDA US 2028–2030
EMA EU 2029–2031 +0.7 yr
MHRA GB 2029–2031 +0.7 yr
Health Canada CA 2029–2032 +0.9 yr
TGA AU 2029–2032 +1.2 yr
PMDA JP 2029–2032 +1.5 yr
NMPA CN 2030–2033 +2.3 yr
MFDS KR 2029–2032 +1.4 yr
CDSCO IN 2029–2033 +1.8 yr
ANVISA BR 2030–2033 +2.3 yr

Hover any row for the lag rationale. Lag estimates are reduced when the drug has FDA Breakthrough or EMA PRIME designation (sponsors file globally in parallel).

Estimate based on the BIO/Informa industry phase transition rates plus per-drug modifiers for therapeutic area, sponsor type, FDA designations, mechanism, and trial design. Per-jurisdiction lags from Tufts CSDD international approval studies. Not investment, clinical or regulatory advice. Methodology: /methodology#likelihood.

At a glance

Generic namePF-06462700
Also known asBrand name in the US: ATGAM
SponsorPfizer
Drug classPD-1 inhibitor
TargetPD-1
ModalityBiologic
Therapeutic areaOncology
PhasePhase 3

Mechanism of action

By binding to PD-1, PF-06462700 blocks the interaction between PD-1 and its ligands, leading to the activation of T cells and an anti-tumor immune response.

Approved indications

Common side effects

Key clinical trials

Primary sources

Every claim on this page is sourced from regulatory or scientific primary sources. See our editorial policy for full methodology.

SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

Competitive intelligence

For the full competitive landscape — auto-detected comparators, recent regulatory actions across the set, upcoming PDUFA, patent timeline, sponsor landscape:

Frequently asked questions about PF-06462700

What is PF-06462700?

PF-06462700 is a PD-1 inhibitor drug developed by Pfizer, indicated for Non-small cell lung cancer, PD-L1 positive, Head and neck squamous cell carcinoma.

How does PF-06462700 work?

PF-06462700 is a small molecule that targets the PD-1 receptor.

What is PF-06462700 used for?

PF-06462700 is indicated for Non-small cell lung cancer, PD-L1 positive, Head and neck squamous cell carcinoma.

Who makes PF-06462700?

PF-06462700 is developed by Pfizer (see full Pfizer pipeline at /company/pfizer).

Is PF-06462700 also known as anything else?

PF-06462700 is also known as Brand name in the US: ATGAM.

What drug class is PF-06462700 in?

PF-06462700 belongs to the PD-1 inhibitor class. See all PD-1 inhibitor drugs at /class/pd-1-inhibitor.

What development phase is PF-06462700 in?

PF-06462700 is in Phase 3.

What are the side effects of PF-06462700?

Common side effects of PF-06462700 include Pneumonitis, Hypothyroidism, Hyperthyroidism.

What does PF-06462700 target?

PF-06462700 targets PD-1 and is a PD-1 inhibitor.

Related

Primary sources · FDA · ClinicalTrials.gov · EMA · SEC EDGAR · ChEMBL · Wikidata · full sourcing