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pegylated interferon a-2a

Foundation for Liver Research · FDA-approved active Small molecule

Pegylated interferon alpha-2a activates innate immune responses by binding to interferon alpha receptors, enhancing antiviral and antiproliferative activity.

Pegylated interferon alpha-2a activates innate immune responses by binding to interferon alpha receptors, enhancing antiviral and antiproliferative activity. Used for Chronic hepatitis C virus infection, Chronic hepatitis B virus infection, Certain hematologic malignancies (e.g., chronic myeloid leukemia, follicular lymphoma).

At a glance

Generic namepegylated interferon a-2a
Also known asPegasys
SponsorFoundation for Liver Research
Drug classInterferon alpha
TargetInterferon alpha receptor (IFNAR)
ModalitySmall molecule
Therapeutic areaImmunology, Virology
PhaseFDA-approved

Mechanism of action

Interferon alpha-2a is a cytokine that binds to type I interferon receptors on cell surfaces, triggering JAK-STAT signaling pathways that upregulate antiviral genes and enhance natural killer cell and macrophage activity. Pegylation extends the drug's half-life by reducing renal clearance and proteolytic degradation. This sustained immune activation is particularly effective against hepatitis C and B viruses, and has antiproliferative effects in certain malignancies.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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