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Oral anticoagulation therapy

China National Center for Cardiovascular Diseases · FDA-approved active Small molecule

Oral anticoagulation therapy prevents blood clot formation by inhibiting coagulation cascade factors, reducing thromboembolism risk.

Oral anticoagulation therapy prevents blood clot formation by inhibiting coagulation cascade factors, reducing thromboembolism risk. Used for Atrial fibrillation for stroke prevention, Venous thromboembolism prevention and treatment, Mechanical heart valve thromboprophylaxis.

At a glance

Generic nameOral anticoagulation therapy
Also known aswarfarin or rivaroxaban, Warfarin/Coumadin, New oral anticoagulants (NOACs): dabigatran, rivaroxaban, apixaban
SponsorChina National Center for Cardiovascular Diseases
Drug classOral anticoagulant
ModalitySmall molecule
Therapeutic areaCardiovascular
PhaseFDA-approved

Mechanism of action

Oral anticoagulants work through multiple mechanisms depending on the specific agent—vitamin K antagonists inhibit factors II, VII, IX, and X, while direct oral anticoagulants (DOACs) directly inhibit factor Xa or thrombin (factor IIa). These mechanisms prevent the formation of fibrin clots and reduce the risk of thrombotic events in conditions like atrial fibrillation and venous thromboembolism.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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