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open-label denosumab

GlaxoSmithKline · Phase 3 active Small molecule Under review

open-label denosumab is a RANKL inhibitor (monoclonal antibody) Small molecule drug developed by GlaxoSmithKline. It is currently in Phase 3 development for Prevention of skeletal-related events in patients with bone metastases from solid tumors, Treatment of osteoporosis in postmenopausal women, Giant cell tumor of bone.

Denosumab is a monoclonal antibody that inhibits RANKL (receptor activator of nuclear factor kappa-B ligand), blocking osteoclast formation and bone resorption.

Denosumab is an open-label study intervention that targets tumor necrosis factor ligand superfamily member 11, an inhibitor of this protein. It is used to treat conditions such as osteoporosis, colorectal cancer, low bone mass, and low bone mineral density, and is administered subcutaneously.

Likelihood of approval
64.3% vs 58.3% industry baseline
If approved by FDA: likely 2028–2030
Steps remaining: NDA/BLA submission
Confidence: High
Why this estimate
  • Baseline phase 3 → approval rate +58.3pp
    Industry-wide phase 3 drugs reach approval ~58.3% of the time (BIO/Informa 2023 industry benchmark across all therapeutic areas).
  • Oncology Phase 3 boost +3.0pp
    Oncology Phase 3 trials have higher approval rates (~61%) than the cross-industry average due to clearer endpoints and FDA oncology pathway.
  • Big-pharma sponsor +3.0pp
    GlaxoSmithKline is a top-20 pharma sponsor — historical approval rates run ~3pp above average due to scale, regulatory experience, and trial-design quality.
Predicted approval windows by jurisdiction (conditional on FDA approval)
Regulator Country Likely year Lag vs FDA
FDA US 2028–2030
EMA EU 2029–2031 +0.7 yr
MHRA GB 2029–2031 +0.7 yr
Health Canada CA 2029–2032 +0.9 yr
TGA AU 2029–2032 +1.2 yr
PMDA JP 2029–2032 +1.5 yr
NMPA CN 2030–2033 +2.3 yr
MFDS KR 2029–2032 +1.4 yr
CDSCO IN 2029–2033 +1.8 yr
ANVISA BR 2030–2033 +2.3 yr

Hover any row for the lag rationale. Lag estimates are reduced when the drug has FDA Breakthrough or EMA PRIME designation (sponsors file globally in parallel).

Estimate based on the BIO/Informa industry phase transition rates plus per-drug modifiers for therapeutic area, sponsor type, FDA designations, mechanism, and trial design. Per-jurisdiction lags from Tufts CSDD international approval studies. Not investment, clinical or regulatory advice. Methodology: /methodology#likelihood.

At a glance

Generic nameopen-label denosumab
SponsorGlaxoSmithKline
Drug classRANKL inhibitor (monoclonal antibody)
TargetRANKL (Receptor Activator of Nuclear Factor Kappa-B Ligand)
ModalitySmall molecule
Therapeutic areaOncology, Bone Metabolism
PhasePhase 3

Mechanism of action

Denosumab binds to RANKL, a key mediator of osteoclast differentiation and activation. By preventing RANKL from engaging its receptor RANK on osteoclast precursors, the drug suppresses bone resorption and increases bone mineral density. This mechanism makes it effective for conditions characterized by excessive bone loss or osteolytic lesions.

Approved indications

Common side effects

Key clinical trials

Primary sources

Every claim on this page is sourced from regulatory or scientific primary sources. See our editorial policy for full methodology.

SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

Competitive intelligence

For the full competitive landscape — auto-detected comparators, recent regulatory actions across the set, upcoming PDUFA, patent timeline, sponsor landscape:

Frequently asked questions about open-label denosumab

What is open-label denosumab?

open-label denosumab is a RANKL inhibitor (monoclonal antibody) drug developed by GlaxoSmithKline, indicated for Prevention of skeletal-related events in patients with bone metastases from solid tumors, Treatment of osteoporosis in postmenopausal women, Giant cell tumor of bone.

How does open-label denosumab work?

Denosumab is a monoclonal antibody that inhibits RANKL (receptor activator of nuclear factor kappa-B ligand), blocking osteoclast formation and bone resorption.

What is open-label denosumab used for?

open-label denosumab is indicated for Prevention of skeletal-related events in patients with bone metastases from solid tumors, Treatment of osteoporosis in postmenopausal women, Giant cell tumor of bone.

Who makes open-label denosumab?

open-label denosumab is developed by GlaxoSmithKline (see full GlaxoSmithKline pipeline at /company/gsk).

What drug class is open-label denosumab in?

open-label denosumab belongs to the RANKL inhibitor (monoclonal antibody) class. See all RANKL inhibitor (monoclonal antibody) drugs at /class/rankl-inhibitor-monoclonal-antibody.

What development phase is open-label denosumab in?

open-label denosumab is in Phase 3.

What are the side effects of open-label denosumab?

Common side effects of open-label denosumab include Hypocalcemia, Osteonecrosis of the jaw, Atypical femoral fractures, Infections, Back pain.

What does open-label denosumab target?

open-label denosumab targets RANKL (Receptor Activator of Nuclear Factor Kappa-B Ligand) and is a RANKL inhibitor (monoclonal antibody).

Related

Primary sources · FDA · ClinicalTrials.gov · EMA · SEC EDGAR · ChEMBL · Wikidata · full sourcing