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Olaparib Treatment D

AstraZeneca · FDA-approved active Small molecule

Olaparib inhibits poly(ADP-ribose) polymerase (PARP) enzymes, preventing DNA repair in cancer cells and causing cell death, particularly in BRCA-mutant tumors.

Olaparib (Lynparza) is a PARP inhibitor developed by AstraZeneca for BRCA-mutant and homologous recombination deficiency (HRD)-positive cancers. The drug works by inhibiting poly(ADP-ribose) polymerase, preventing DNA repair in tumors with defective homologous recombination pathways, leading to synthetic lethality. Approved indications span ovarian cancer (platinum-sensitive and resistant), breast cancer, pancreatic cancer, and prostate cancer across multiple treatment lines. Olaparib is a first-in-class PARP inhibitor and remains a cornerstone of precision oncology, with peak annual sales exceeding $1.5B globally. The drug has demonstrated clinical differentiation through both monotherapy and combination regimens (notably with bevacizumab in ovarian cancer), and ongoing trials explore expansion into additional solid tumors including bladder, lung, and adrenal cancers. Commercial significance is substantial given the large addressable market in BRCA-positive populations and the shift toward biomarker-driven treatment paradigms.

At a glance

Generic nameOlaparib Treatment D
SponsorAstraZeneca
Drug classPARP inhibitor
TargetPARP1, PARP2
ModalitySmall molecule
Therapeutic areaOncology
PhaseFDA-approved

Mechanism of action

PARP inhibitors block the repair of single-strand DNA breaks. In BRCA1/2-deficient cancers that lack homologous recombination repair, this leads to accumulation of DNA damage and synthetic lethality. Olaparib is effective as monotherapy in BRCA-mutant cancers and as maintenance therapy following chemotherapy in platinum-sensitive ovarian cancer.

Approved indications

Common side effects

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