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NSAID's

Saglik Bilimleri Universitesi Gulhane Tip Fakultesi · FDA-approved active Small molecule

NSAIDs inhibit cyclooxygenase (COX) enzymes to reduce prostaglandin synthesis, thereby decreasing inflammation, pain, and fever.

NSAIDs inhibit cyclooxygenase (COX) enzymes to reduce prostaglandin synthesis, thereby decreasing inflammation, pain, and fever. Used for Mild to moderate pain, Fever, Inflammatory conditions (e.g., rheumatoid arthritis, osteoarthritis).

At a glance

Generic nameNSAID's
Also known asDiclophenac Potassium
SponsorSaglik Bilimleri Universitesi Gulhane Tip Fakultesi
Drug classNon-steroidal anti-inflammatory drug (NSAID)
TargetCyclooxygenase-1 (COX-1) and Cyclooxygenase-2 (COX-2)
ModalitySmall molecule
Therapeutic areaPain management, Inflammation, Rheumatology
PhaseFDA-approved

Mechanism of action

NSAIDs block COX-1 and/or COX-2 enzymes, which catalyze the conversion of arachidonic acid to prostaglandins and thromboxanes. By reducing prostaglandin levels, NSAIDs suppress inflammatory mediators, reduce pain signaling, and lower fever set points. Different NSAIDs vary in their selectivity for COX-1 versus COX-2, affecting their efficacy and side effect profiles.

Approved indications

Common side effects

Key clinical trials

Primary sources

Every claim on this page is sourced from regulatory or scientific primary sources. See our editorial policy for full methodology.

SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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