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MYL-1701P, a proposed biosimilar to Eylea

Mylan Pharmaceuticals Inc · Phase 3 active Biologic

MYL-1701P is a biosimilar to aflibercept that binds vascular endothelial growth factor (VEGF) and placental growth factor (PlGF) to inhibit pathological neovascularization in the eye.

MYL-1701P is a biosimilar to aflibercept that binds vascular endothelial growth factor (VEGF) and placental growth factor (PlGF) to inhibit pathological neovascularization in the eye. Used for Neovascular (wet) age-related macular degeneration, Retinal vein occlusion, Diabetic macular edema.

At a glance

Generic nameMYL-1701P, a proposed biosimilar to Eylea
SponsorMylan Pharmaceuticals Inc
Drug classMonoclonal antibody (fusion protein) / VEGF inhibitor / Biosimilar
TargetVEGF-A, VEGF-B, PlGF (vascular endothelial growth factor and placental growth factor)
ModalityBiologic
Therapeutic areaOphthalmology
PhasePhase 3

Mechanism of action

As a biosimilar to Eylea (aflibercept), MYL-1701P is a recombinant fusion protein that acts as a VEGF trap, sequestering VEGF-A, VEGF-B, and PlGF to prevent their interaction with endothelial growth factor receptors. This mechanism reduces abnormal blood vessel growth and vascular permeability in retinal diseases. The biosimilar is designed to have comparable efficacy and safety to the reference product in treating neovascular age-related macular degeneration and other retinal conditions.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results

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