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Mavenclad®

Merck Healthcare KGaA, Darmstadt, Germany, an affiliate of Merck KGaA, Darmstadt, Germany · FDA-approved active Small molecule

Mavenclad (cladribine) is a purine nucleoside analog that depletes lymphocytes by interfering with DNA synthesis and repair in dividing cells.

Mavenclad (cladribine) is a purine nucleoside analog that selectively depletes lymphocytes, particularly CD4+ and CD8+ T cells and B cells, to reduce autoimmune activity. Used for Relapsing-remitting multiple sclerosis (RRMS), Active secondary progressive multiple sclerosis (SPMS).

At a glance

Generic nameMavenclad®
Also known asCladribine
SponsorMerck Healthcare KGaA, Darmstadt, Germany, an affiliate of Merck KGaA, Darmstadt, Germany
Drug classPurine nucleoside analog; immunosuppressant
TargetDeoxyadenosine kinase; DNA synthesis and repair pathways in lymphocytes
ModalitySmall molecule
Therapeutic areaImmunology; Neurology
PhaseFDA-approved

Mechanism of action

Cladribine is a deoxyadenosine analog that accumulates in lymphocytes and is phosphorylated to its active triphosphate form, which inhibits ribonucleotide reductase and disrupts DNA synthesis. This leads to selective depletion of B and T lymphocytes, reducing the autoimmune response. The drug has prolonged effects due to sustained lymphocyte depletion following short treatment courses.

Approved indications

Common side effects

Key clinical trials

Primary sources

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SourceUsed for
ClinicalTrials.govTrial enrolment, design, endpoints, results